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نیازمندی ها ...!
ســــــــــــــلام 
میشه لطفا هر کی چند تا وبلاگ که از نظر خودش خوبه رو به من معرفی کنه !؟! (غیر از وبلاگ خودش!)
هر چی بیشتر بهتر.
خسته شدم از بس گشتم ...!
باتشکر ! 
من...!
چهارشنبه 31 فروردین 1390 ساعت 14:25
خب هر کس سلیقه های متفاوتی داره، کاشکی میگفتی که چه جور وبلاگی دوست داری که بقیه بتونن معرفی کنن
خب هر کی به سلیقه خودش معرفی کنه. من میرم میبینم اگه خوشم اومد که خوبه اگه نیومد هم که هیچی دیگه...
!
سلام.
خوبی...
ازت ممنونم بابت همه حرفایی که زدی.با این که با همشون موافق نبودم ولی دلگرم کننده بودن.
و این بهترین چیز واسه منه...
امیدوارم بتونم جبران کنم...
______________________
سلام
حالا ماه شدی ! (تبلیغ کرم ببک :دی)
خیلی خوشحال شدم که دوباره برگشتی.
سلام. این وبلاگ الان پیدا شد. شاعرانه ست:
http://lore.blogsky.com
آفریــــــــــن ! مرسی برم ببینم چی هست.....
ممنون
اولا این یکی وبلاگمم ببین .
دوما خیلی مرسی که اومدی وبلاگم . خو.شحالم کردی امیدوارم بیشترببینمت. این یکی وبلاگم یه سری داستان طنزه امید وارم خوشت بیاد .
سه تا وبلاگی رو که خودم دوست دارم رو بهت معرفی میکنم
از مجید گلم برای این که وبلاگم رو بهت توصیه کرده ممنونم
http://anidalton.blogfa.com/
http://www.iran-old-song-2.blogsky.com/
http://bfnadaram.blogsky.com/#
ســــلام دوست جدید من !
آری آری!! من از مجید خیلی ممنونم 
واقعا کلی هیجان زده شدم !!
باشه اینم میبینم
مرسی اومدی
مرسی واسه این وبلاگ ها که معرفی کردی
مرســـی مرســـــــــــی
سلام عسیسم.
خوفی؟
من یه وبلاگ هست که خیلی از خوندش لذت می‌برم، آدرسش :
http://porpot.blogsky.com
گفتم بگم شاید تو هم دوست داشته باشی.
بوس بوس.
مدیونی اگه فکر کنی من خودمم
خب من با غریبه ها حرف نمیزنم!!
واقعا که! یعنی دخترا اینجوری حرف می زنن دیگه؟!!
خب حالا من چی کار کنم که مدیون شدم؟!!
حالا کی گوسفند رو بکشیم واسه اینکه شما تشریف آوردید اینجا؟!!
سلام خانموی
مرسی که میای و سر میزنی و خوشحالم میکنی
واما وبلاگ::
http://bighazal.blogfa.com/9001.aspx
http://anti-2khtar.blogsky.com
http://vyonna.blogfa.com/
http://anidalton.blogfa.com
http://saba055.blogsky.com/
http://www.birag.blogsky.com/
http://reminders.blogsky.com/
امیدوارم خوشت بیاد
بازم برات میسندم
(گفتم هر روز بودی ها چرا نیستی .... )
ممنــــــــــون واسه این همه وبلاگی که معرفی کردی
وای منم هستم که!! :دی
یه سایت خوب :
http://charismaco.com/html/
الان اینا رو جواب بدم میرم میبینم!!
http://gfnadaram.blogsky.com/
وااای بازم مرسی !
مرسی که یادت بود...!
سلام
بازم اومدم
http://deleasemooni.blogsky.com/
http://zane27sale.blogfa.com/
خوش اومدی
منو شرمنده میکنیا ! :دی
کلی ممنون
بچه ها
خیلی خیلـــــی خیلی .... ممنـــــــــــــونم از همتون
خواهش
یه وبلاگ شعر هست واسه شاعر مرود علاقه منه داستانشم میذاره
امیدوم از اینم خوشت بیاد
www.sybryae.blogspot.com
البته بگما بلاگ اسپوت فیلتره با فیلتر شکن برو بخون
چشم !!
سلام عنوان وبلاگو عوض کردم بدو نظرتو بهم بگو
خوب کاری کردی!
شرمنده ها من از دوشنبه دارم میدوم امروز رسیدم! یه کم مسافت زیاد بود!!
سلام
وب جالبی داری
به منم سر بزن..نظراتم بگو.خوشحال میشم
چه باحال! این یکی خودش خودشو معرفی کرد!!
سلام! باشه میام! :دی
سلام چه کار جالبی کردی این فراخوان را دادی
ولی من می خوام فقط ادرس خودم را بهت بدم چون همه ی ادرس های خوبم را اونجا لینک می کنم .
به هر حال نمی شه به همیشگی بودن بوکمارک ها و لیست فیبوریتس دل بست.
سلام :) ایده جالبی بود ممنون عزیزم
سلام خوبی
این وبلاگ خیلی حرفا داره ادمو به اندیشه وا میداره اگه متوجه شی
وبلاگ گفتگوی من با خدا توگوگل سرچش کن
khoda19.blogfa.com
Agomelatine does not appear to cause sexual side effects or weight gain, but may elevate serum transaminase levels more than three times the upper limit of normal.
(Uptodate)
Asenapine and Clozapine can be considered for patients who do not respond to first-line treatments for acute mania or mixed states. In randomized trials, Asenapine 5 to 10 mg two times per day was more efficacious than placebo, but much less efficacious than olanzapine 5 to 20 mg once per day. Common side effects of Asenapine include sedation, fatigue, dizziness, extrapyramidal symptoms, vomiting, dry mouth, and weight gain.
Bipolar disorder: Maintenance treatment Asenapine — Asenapine is prescribed at a dose of 5 to 10 mg twice per day. Common side effects include weight gain, sedation, dizziness, insomnia, and extrapyramidal symptoms. A short-term study suggests asenapine prevents early relapse of mood episodes as well as olanzapine. Patients who completed a three-week randomized trial comparing asenapine versus olanzapine for treatment of acute manic or mixed episodes were enrolled in a nine-week extension study if they wished to continue treatment. Scores on various rating scales revealed no significant difference between the two drugs. (Uptodate)
Azelnidipine: Calcium Channel Blocker, for Management of hypertension. Tablet: 8 mg, 16 mg
Bazedoxifene — Bazedoxifene is effective in preventing and treating osteoporosis. As examples:
In a two-year randomized trial of bazedoxifene, raloxifene, or placebo in 1583 postmenopausal women at risk for osteoporosis (T-score of the lumbar spine or femoral neck between -1.0 and -2.5 or other risk factors), women randomly assigned to bazedoxifene or raloxifene similarly maintained spine and hip BMD, whereas women in the placebo group had significant bone loss at both sites (between group differences of approximately 1.5 and 1.6 percent, respectively).In a three-year randomized trial in 6847 postmenopausal women with osteoporosis, the cumulative incidence of new vertebral fractures was significantly lower in women randomly assigned to bazedoxifene (20 or 40 mg daily) or raloxifene (60 mg daily) versus placebo (fracture rates of 2.3, 2.5, 2.3 versus 4.1 percent).
In both trials, hot flashes and leg cramps were the most common adverse events, occurring in up to 24 and 12 percent of subjects, respectively. The rates of endometrial hyperplasia, cancer, and polyps were low and similar among the groups. In one of the trials, 753 of 6847 women underwent periodic transvaginal ultrasonography to assess endometrial safety and 444 had mammography to assess breast density. After two years, the following findings were noted:
There were no between group differences in mean endometrial thickness (2.26, 2.51, 2.56, and 2.26 mm in the bazedoxifene 20 mg, bazedoxifene 40 mg, raloxifene 60 mg, and placebo groups, respectively) or mean change from baseline in endometrial thickness (-0.07, 0.10, 0.16, and -0.08 mm, respectively).The incidence of ovarian cancer was low. There was evidence of a significant increase in the incidence of ovarian cancer with bazedoxifene 20 mg (five cases). However, there were no cases with bazedoxifene 40 mg, which seems to make a causal relationship unlikely.Changes in breast density were low and not significantly different among the four groups.
Bazedoxifene/conjugated estrogen_A combination of bazedoxifene and conjugated estrogen (BZA/CE) is under investigation for the treatment of postmenopausal vasomotor symptoms and osteoporosis prevention
Dapoxetine
An additional SSRI, dapoxetine, also appears to be effective, based upon two randomized trials of nearly 2000 men with premature ejaculation who were randomly assigned to receive placebo or dapoxetine (30 mg or 60 mg/day) [89]. Unlike other SSRIs, which are taken daily, dapoxetine is taken on-demand one to three hours before intercourse. The mean ejaculatory latency time before treatment in all groups was approximately 0.9 minutes, while the mean intervals in the placebo and dapoxetine groups (30 and 60 mg) on treatment were 1.8, 2.8, and 3.3 minutes, respectively. The most common side effect with dapoxetine was nausea (8 and 20 percent with 30 and 60 mg, respectively).
Desvenlafaxine
Antidepressant, SNRIs.
Tablet, 50 mg, 100 mg
Desvenlafaxine, the main active metabolite of venlafaxine, was approved for use as an antidepressant in the United States in 2008. Desvenlafaxine is the main active metabolite of venlafaxine, and their side effect profiles appears similar.
Desvenlafaxine, unlike venlafaxine, is not processed through the CYP2D6 metabolic pathway, so inhibitors of CYP2D6 should have no effect on drug plasma levels. It is metabolized in the liver by direct conjugation and, to a lesser degree, through CYP3A4.
Desvenlafaxine is formulated for once daily dosing, and is approved for depression treatment at a dose of 50 mg daily. There is no data showing improved efficacy for the 100 mg dose which is also available.
There is no compelling reason to prescribe desvenlafaxine instead of the extended-release formulation of venlafaxine (venlafaxine XR), particularly since generic availability of venlafaxine XR is anticipated in 2010 and it should then be significantly less expensive than desvenlafaxine.
Dronedarone
Antiarrhythmic Agent, Class III
Tablet, oral: Multaq®: 400 mg
Dronedarone and Severe Liver Injury:
The U.S. Food and Drug Administration (FDA) is notifying healthcare professionals and patients about rare reports of dronedarone (Multaq®) causing severe liver injury, including acute liver failure leading to liver transplant in two patients. Information regarding the potential risk of liver injury is being added to the labeling of dronedarone (Warnings, Precautions, and Adverse Reactions sections).
Healthcare professionals should consider periodically monitoring serum liver enzymes and bilirubin, especially during the first 6 months of therapy. If liver injury is suspected, dronedarone therapy should be discontinued and liver enzymes and bilirubin should be measured. Appropriate treatment should be started and dronedarone therapy should not be reinitiated if liver injury is confirmed. Patients should be advised to discuss any signs or symptoms of hepatic injury with a healthcare professional.
Dronedarone has a very low proarrhythmic risk like amiodarone, but is not as effective as amiodarone in maintaining sinus rhythm.
Dronedarone, like amiodarone, is a multichannel blocker that prolongs with repolarization.
Dronedarone — Dronedarone is a derivative of amiodarone that lacks the iodine moieties, but has a methylsulfonamide group. This structural change not only theoretically reduces systemic side effects, but also makes the analogue less lipophilic and thus decreases the volume of distribution. It is believed that the iodine moieties on the molecule play a major role in mediating these systemic side effects. However, the iodine may contribute to the efficacy of the molecule as well. As a result, the elimination half-life is dramatically reduced to approximately one day with a reduction in neurologic, thyroid, liver, and lung toxicity. However, most recently there are reports that dronedarone may be linked to liver injury in patients and thus the FDA has suggested monitoring liver enzymes. (See "Major side effects of amiodarone".)
In patients with AF, randomized trials with up to 12 months of follow-up have found that dronedarone prevents recurrent AF and was safe (including no increased risk of serious arrhythmias). However, in the ANDROMEDA trial in patients with advanced heart failure from LV systolic dysfunction, there was an increased risk of death with dronedarone and the trial was stopped early. As a result, dronedarone is contraindicated in this population of patients. (See "Atrial fibrillation in patients with heart failure", section on 'Antiarrhythmic drug therapy'.)
The ATHENA trial was a placebo-controlled randomized trial of 4628 patients that examined whether dronedarone reduces the rate of death or hospitalization due to cardiovascular events in patients with persistent or paroxysmal AF and additional risk factors for death. Patients with NYHA class II or III heart failure comprised 21 percent of the study population. An important difference between ATHENA and ANDROMEDA is that patients with decompensated or NYHA class IV heart failure were excluded in ATHENA. In addition, it is important to note that maintenance of sinus rhythm was not one of the endpoints in ATHENA.
After a mean follow-up period of 21 months, dronedarone significantly reduced the primary outcome of death or cardiovascular hospitalization (31.9 versus 39.4 percent, hazard ratio 0.76, 95% CI 0.69-0.84) and the secondary outcome of cardiovascular death (2.7 versus 3.9 percent, hazard ratio 0.71, 95% CI 0.51-0.98). Dronedarone is the only antiarrhythmic drug that has shown a salutary effect on mortality.
There have been two case reports of severe hepatotoxicity with a need for liver transplantation in patients taking dronedarone.
The DIONYSOS study was a short-term (median duration of seven months) comparison between amiodarone and dronedarone to assess the differences in drug tolerability and AF recurrence in 504 patients. Sixty percent of the patients had persistent AF. The authors found that the composite primary endpoint of AF recurrence or premature study drug discontinuation occurred in 75.5 percent of patients taking dronedarone, but only 58.8 percent of patients taking amiodarone. This endpoint was primarily driven by AF recurrence on dronedarone compared to amiodarone (63.5 versus 42.0 percent, respectively). Drug discontinuation and the main safety endpoints of extra-cardiac toxicity only tended to be less with dronedarone, but did not reach statistical significance. It is possibly that with longer follow-up periods there would have been a greater difference in noncardiac side effects, since the toxicity with amiodarone is typically manifest after several months to years of use. A meta-analysis of dronedarone trials prior to the DIONYSOS study where the effect of amiodarone versus dronedarone was estimated with the use of indirect comparison and normal logistic meta-analysis models, a similar conclusion was reached [77]. Amiodarone was found to be more effective in maintaining sinus rhythm, but at the expense of greater drug discontinuation secondary to adverse events.
Febuxostat
Uloric®: 40 mg, 80 mg
Xanthine Oxidase Inhibitor
Mechanism — Febuxostat is a xanthine oxidase inhibitor. It is a thiazolecarboxylic acid derivative that, unlike allopurinol, is not a purine base analogue.Efficacy — Febuxostat produces a dose-dependent decrease in serum urate levels [59]. A daily dose of 40 mg produces a reduction that is roughly equivalent to that seen in patients who are treated with allopurinol at a dose of 300 mg per day [46,60].
A phase III 52-week trial involved 760 patients with gout and serum urate ≥8 mg/dL [≥476 micromol/L] who were assigned to febuxostat at daily doses of 80 or 120 mg or to allopurinol at 300 mg daily [46]. Although serum urate levels were significantly reduced in all three study groups, the primary efficacy end point of the study, reduction of serum urate to a target level <6 mg/dL [<357 micromol/L] on each of the last three study visits, was achieved by a significantly greater proportion of those in the in the 80 mg febuxostat and 120 mg febuxostat groups than those in the allopurinol group (53 and 62 versus 21 percent).
Trends toward reductions in gout flare incidence and in tophus areas over time were observed in all three treatment groups, but these end points were not significantly different among the groups over the year-long trial. The incidence of adverse events did not differ across the groups, but withdrawal from the study due to gout flares and abnormal liver function studies was more common in both febuxostat groups than the allopurinol group.
In a second phase III safety and efficacy trial of six months' duration [61], febuxostat (at daily doses of 80 mg or 120 mg) was compared with allopurinol (300 mg, or, in the case of subjects with renal functional impairment, 100 mg) and placebo. This study confirmed the superiority in urate-lowering efficacy of febuxostat at both doses relative to allopurinol and placebo, both in subjects with normal renal function and in a small subgroup of subjects with mild to moderate CKD. No dose reduction in febuxostat appeared necessary in subjects with these levels of CKD.
The use of febuxostat in patients with mild or moderate renal functional impairment (defined, respectively, as estimated creatinine clearance of 60 to 89 mL/min and 30 to 59 mL/min) was assessed as part of a six-month study that randomly assigned 2268 subjects with gout and baseline sUA ≥8 mg/dL (476 micromol/L) to receive febuxostat 40 mg or 80 mg daily or allopurinol (300 mg daily in subjects with normal or mildly impaired renal function or 200 mg daily if baseline renal function was moderately impaired) [60]. Approximately 65 percent of subjects in each treatment group had either mild or moderate renal impairment, and in this subset of patients, urate-lowering with febuxostat at either dose was more likely to achieve the serum urate goal. Safety was comparable across all treatment groups.
Dose — Febuxostat received approval from the US Food and Drug Administration for treatment of hyperuricemia in gout at daily doses of 40 mg and 80 mg [62]. The American manufacturers recommended starting dose is 40 mg once daily. An increase in dose to 80 mg daily is suggested for patients whose serum urate level does not fall to <6 mg/dL (<357 micromol/L) after two weeks of treatment on the lower (40 mg daily) dose. In Europe higher doses have received regulatory approval (80 to 120 mg/day) [63].Important drug interactions — Decreased metabolism of azathioprine, mercaptopurine, and theophylline are expected results of administration of a xanthine oxidase inhibitor and a need for continued use of any of these three drugs is considered by the manufacturer a contraindication to the use of febuxostat [64]. As noted above, we and others have used reduced doses of azathioprine or mercaptopurine with the xanthine oxidase inhibitor allopurinol while carefully monitoring for bone marrow toxicity (ie, development of cytopenias). It is uncertain at present whether such a strategy can safely be pursued with febuxostat.Side effects — A greater incidence of liver function test abnormalities, nausea, arthralgia, and rash have been noted in febuxostat-treated patients than placebo controls, but not allopurinol-treated subjects, during clinical trials. Periodic monitoring of liver function, principally hepatic transaminase enzyme levels, is suggested by the manufacturer of febuxostat [64].
During clinical trials, patients treated with febuxostat had a higher incidence of cardiovascular events than controls who received allopurinol (0.74 [95% CI 0.36-1.37] versus 0.60 [95% CI 0.16-1.53] events per 100 patient years, respectively) [64]. Although such an imbalance was not confirmed in the most recently published randomized trial [60], a postmarketing study relevant to this finding is in progress.
As with any urate lowering treatment, the risk of acute gout attacks is increased when febuxostat is initiated. Prophylaxis for acute attacks is recommended (see 'Prophylaxis during initiation of antihyperuricemic therapy' below).Cost — The cost of treatment with febuxostat is substantially higher than with either generic allopurinol or its brand name equivalent. The average retail cost of a 30-day supply of febuxostat in retail pharmacies in the United States is reported to be 161.4 dollars, more than ten times the cost of the generic allopurinol (300 mg/day) and four times as much as the Promethesus pharmaceutical brand of allopurinol.
Flupirtine
Two other trials evaluated flupirtine and tolperisone, which are not available in the US. Both medications were more effective than placebo.
Flupirtine — Flupirtine maleate is a centrally acting, nonopioid analgesic that has displayed cytoprotective activity in vitro in neurons inoculated with a prion protein fragment [51]. The mechanism of neuroprotective action is unknown, but it may involve up-regulation of the anti-apoptotic protein bcl-2. Alternatively, its N-methyl-D-aspartate antagonist properties might limit glutamate-mediated neurotoxicity.
Flupirtine is not currently available in the United States. In a European study, 28 patients with CJD were randomly assigned to treatment with flupirtine (n=13) or placebo (n = 15). One patient in each group had familial CJD, the remainder were sporadic cases. Diagnosis was based upon the World Health Organization (WHO) criteria for "probable" CJD. Flupirtine treatment was initiated at 100 mg per day, and increased over three days to a maintenance dose of 300 to 400 mg per day, which was continued for a median treatment duration of 29 days. There was no significant effect of flupirtine treatment on survival time compared with placebo. However, the patients treated with flupirtine performed significantly better on the cognitive part of the Alzheimer Disease Assessment Scale (ADAS-Cog). Flupirtine-treated patients also did better on the Mini Mental Status Examination, but the difference did not reach statistical significance. Caregiver's impressions were also significantly better in the flupirtine-treated group.
Iloperidone
Tablet 1 mg, 2 mg, 4 mg, 6 mg, 8 mg, 10 mg, 12 mg
Antipsychotic Agent, Atypical
Iloperidone (Fanapt™) — Iloperidone is the most recently approved antipsychotic in the US. Clinical experience is limited. Efficacy has been demonstrated for short- and long-term treatment of schizophrenia [52,53]. It has antagonist activity at dopamine D2 and serotonin 5HT2a receptors, similar to other atypicals. Iloperidone was relatively well tolerated in short-term studies, with a discontinuation rate similar to placebo.
Side effects include dizziness, orthostatic hypotension, tachycardia, weight gain, dry mouth, and sedation [54,55]. Compared to haloperidol or risperidone, iloperidone caused fewer extrapyramidal symptoms but slightly more qT prolongation; weight gain was intermediate.
Short-term studies found optimal efficacy at 20 to 24 mg daily [52] and the drug is approved at doses of 12 to 24 mg daily.. Because of the propensity for orthostatic hypotension, dosing should be initiated at 1 mg bid with gradual titration. The elimination half-time is 18 to 33 hours. Plasma levels are sensitive to CYP 2D6 inhibitors such as fluoxetine.
Few patients over 65 years of age were included in clinical trials, so specific information on dosing and side effects in the older population is not available. The known risks of hypotension and qT prolongation suggest that caution should be exercised in the use of iloperidone in older patients.
Lafutidine
Tablet: 5 mg, 10 mg
Histamine H2 Antagonist
Treatment of gastric, duodenal, and stomal ulcers; acute gastritis and acute exacerbation of chronic gastritis; preanesthetic
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